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Glycine Methionine Balance Revisited: A Matter of Timing

By Joel Brind In one of my earlier posts on this blog, I discussed the largely reciprocal nature of the amino acids glycine and methionine. Specifically, too much dietary methionine depletes glycine, because your body uses up glycine in order to get rid of the excess methionine. This is a common condition these days, because the typical diet is high in methionine-rich muscle meats, but low in...

Good Proteins, Bad Proteins: The Amino Acids in Health and Disease

Good Proteins, Bad Proteins… 1. Preface: Not All Protein Is Created Equal A few months ago, an interesting Danish study was published, showing that a?”high-fat/high-sugar” diet?would cause obesity to mice consuming chicken, cod, or crab as their main protein source. However, the group of mice eating scallop protein didn’t develop these problems at all. The researchers...

Glutathione: Is More Better?

By Joel Brind, Ph.D. The subject of glutathione (GSH) came up in my last post on diet and inflammation, some of the comments reflecting the popularity of GSH as some sort of miracle molecule among alternative health purveyors and practitioners. Among the commenters, Sarah said: “Is this total BS? Is glutathione really the miracle substance some alternative med doctors say that it...

Glycine and Cancer

Comment from Martin Tornberg: “One thing that gives me pause with respect to consuming too much glycine is the study on?http://www.sciencemag.org/content/336/6084/1040.abstract’that ?identified glycine consumption and expression of the mitochondrial glycine biosynthetic pathway as strongly correlated with rates of proliferation across cancer cells? and noted that ?Antagonizing...

Diet and Inflammation Part 4

By Joel Brind, Ph.D. My last post focused on the propagation of the inflammatory signal, and how nutrients such as omega-6 PUFAs and the lack of salicylic acid results in amplification of that signal, thus contributing to the overall increase in inflammation-related disease we currently experience. But what about the initiation of the inflammatory signal in the first place? That often turns out...